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Strength sport places unique demands on kidney, liver, and hormonal systems. This panel keeps all three under informed review.
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Tailored for weightlifters, powerlifters, and physique athletes. Covers anabolic hormones, kidney and liver function, lipid profile, and haematology.
Strength and power sports require a different physiological monitoring strategy than endurance events. High-volume resistance training, significant caloric intake, and (in some individuals) use of performance-influencing supplements place specific stress on the kidneys, liver, and cardiovascular system. At the same time, the anabolic hormone axis — testosterone, SHBG, LH, and IGF-1 — is central to training adaptation. This panel addresses all of these concerns in one comprehensive draw. Kidney function is assessed via creatinine and eGFR (with context that heavily muscled individuals have higher baseline creatinine); liver integrity via ALT and GGT; cardiovascular risk via the full lipid profile; and anabolic hormone status via testosterone, SHBG, and IGF-1. Haemoglobin and haematocrit are included because some performance compounds affect red cell production. A GMC-registered physician reviews every result.
Understand what each marker measures, why it matters, and what the science says — not just a list of numbers.
This panel is designed for adults who want a comprehensive, evidence-based picture of their metabolic health — not a GP referral panel.
Competitive powerlifters, Olympic weightlifters, and strongman athletes
Natural bodybuilders preparing for or recovering from competition
Recreational gym-goers on significant supplementation stacks
Anyone who has used or is considering performance-enhancing compounds and wants objective health monitoring
Personal trainers seeking objective physiological data for client health reviews
Creatinine and eGFR in heavily muscled individuals should be interpreted cautiously; high muscle mass produces more creatinine as a waste product of phosphocreatine metabolism, meaning creatinine may be above the reference range without any renal impairment. Creatine supplementation also elevates serum creatinine independently of muscle mass. ALT rises physiologically in the 24 to 72 hours following intense resistance training and does not necessarily indicate liver pathology; collection should be 48 hours after training for a resting baseline. This panel does not include full hepatitis screen, haematological malignancy markers, or specialist endocrinology testing.
From order to physician-reviewed report in as little as three working days.
Venous draw for the full 15-marker panel. Book online.
48 to 72 hours after last training session. Morning, fasted 10 hours.
Within 24 hours of receipt.
Physician commentary in 3 to 5 working days.
Three options designed to fit your schedule, location, and preference — all producing a laboratory-standard sample.
Adults 18+ in mainland UK. Not suitable if you have had a transfusion in the last 3 months.
Order anytime; kit dispatched within 24 hours Mon–Fri.
Allow 24–48 hours for sample transit on top of lab processing time.
Adults 18+ within 20 miles of a serviced city centre.
Mon–Sun, 06:00–20:00. Next-day booking typical.
Sample reaches the lab within 24 hours of collection.
Adults 16+ with photo ID. Paediatric draws by appointment at selected sites.
Mon–Fri, with Saturday hours at most metropolitan locations.
Samples processed same-day at the receiving clinic.
Every test is processed in a UKAS ISO 15189-accredited laboratory, overseen by GMC-registered physicians, and governed by UK GDPR. No overseas processing, no offshore data.
Follow these guidelines to ensure accurate, reproducible results. Most markers are sensitive to recent food, exercise, and sleep.
Can't find your answer? Our clinical support team is available Monday to Friday, 9am–5pm.
Contact supporteGFR is calculated from serum creatinine, which is a byproduct of creatine metabolism in muscle tissue. Athletes with substantially greater muscle mass produce proportionally more creatinine, pushing serum creatinine higher than sedentary reference populations would expect. Additionally, creatine supplementation — widely used in strength sports — further elevates serum creatinine. The net effect is that eGFR calculated using standard population equations may appear low even in athletes with completely healthy kidneys. Your physician commentary will flag this and contextualise your result relative to your training status.
ALT (alanine aminotransferase) is found in both hepatocytes (liver cells) and muscle tissue. Intensive resistance training, particularly eccentric-heavy sessions, causes muscle fibre micro-tears that release ALT into the bloodstream. This can produce ALT elevations of two to five times the upper limit of the reference range within 24 to 48 hours of training, entirely without liver involvement. To avoid this confounding effect, we ask you to collect at least 48 hours after your last hard training session. If ALT remains elevated at rest, liver-specific markers such as GGT and ALP alongside AST can help distinguish muscle from liver origin.
Regular resistance training generally has a favourable effect on the lipid profile: it raises HDL, lowers triglycerides, and tends to reduce LDL particle size toward a less atherogenic pattern. However, significant caloric surplus diets (commonly used in mass phases) can raise LDL and triglycerides. Some performance-influencing supplements are associated with adverse lipid shifts, particularly lowered HDL. Monitoring your lipid profile provides an early warning system and an objective record of cardiovascular risk alongside your other health markers.
Suppressed testosterone in a strength athlete can stem from several causes: overtraining syndrome, relative energy deficiency (consuming insufficient calories for your training volume), stress and poor sleep, or use of exogenous androgens that suppress the hypothalamic-pituitary-testicular axis. The accompanying LH measurement helps distinguish between primary gonadal insufficiency and central (hypothalamic-pituitary) suppression. Your physician commentary will identify the likely pattern and recommend appropriate next steps, which typically involve lifestyle modification or a GP referral for further evaluation.
Yes. Well-trained natural athletes can have testosterone in the upper half or toward the upper end of the male reference range, particularly if sleep, nutrition, and training periodisation are optimised. High-normal testosterone is associated with faster recovery, greater muscle protein synthesis rates, and better body composition regulation. This is a feature of good training hygiene rather than a pathological finding. The concern arises when testosterone is suppressed below mid-range or when LH is simultaneously very low, suggesting central axis suppression.
