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Your biology tells you where you are headed years before symptoms do. This panel maps your current trajectory.
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A preventative health screen covering metabolic, cardiovascular, inflammatory, and hormonal markers associated with healthy ageing. Home kit available.
Longevity science has moved well beyond the idea that ageing is simply the passage of time. Metabolic dysregulation, chronic low-grade inflammation, cardiovascular risk accumulation, and hormonal decline are the biological processes that drive most age-related disease — and all of them are measurable decades before symptoms emerge. This essential panel screens the key markers of metabolic health (fasting glucose, HbA1c, insulin resistance, lipid profile), inflammation (hs-CRP), hormonal status (testosterone or oestradiol, dehydroepiandrosterone sulphate (DHEA-S)), and organ function (liver, kidney), giving you a comprehensive picture of your current biological trajectory. The goal is not just to extend years but to optimise the quality of those years by identifying and addressing modifiable risk factors while there is still time to intervene effectively. All results include GMC-registered physician commentary.
Understand what each marker measures, why it matters, and what the science says — not just a list of numbers.
This panel is designed for adults who want a comprehensive, evidence-based picture of their metabolic health — not a GP referral panel.
Adults aged 35 and over interested in preventative health monitoring
Anyone with a family history of cardiovascular disease, diabetes, or dementia wanting to assess their own risk
People who prioritise data-driven health decisions and want a comprehensive annual baseline
Biohackers and longevity enthusiasts wanting to track biological age markers
Those who have made lifestyle changes and want objective confirmation of improvement
This panel provides a comprehensive preventative health snapshot but is not a substitute for a full NHS health check or GP consultation. Lp(a) is a genetically determined lipoprotein; elevated levels cannot be modified by diet or standard lipid-lowering therapy and require specialist cardiovascular review. HOMA-IR is a calculated estimate of insulin resistance using fasting glucose and insulin and may be less accurate in extreme obesity or liver disease. dehydroepiandrosterone sulphate (DHEA-S) declines with age but the reference range is broad; a single result should be interpreted relative to your age-adjusted range, not the full population range. This panel does not screen for cancer, assess cognitive function, or evaluate arterial stiffness or other physical markers of biological age.
From order to physician-reviewed report in as little as three working days.
Home fingerstick kit or mobile phlebotomist for fasting venous draw.
10-hour fast required for glucose, insulin, and lipid accuracy.
Pre-paid Royal Mail envelope included; same-day despatch.
Comprehensive physician commentary in 3 to 5 working days.
Three options designed to fit your schedule, location, and preference — all producing a laboratory-standard sample.
Adults 18+ in mainland UK. Not suitable if you have had a transfusion in the last 3 months.
Order anytime; kit dispatched within 24 hours Mon–Fri.
Allow 24–48 hours for sample transit on top of lab processing time.
Adults 18+ within 20 miles of a serviced city centre.
Mon–Sun, 06:00–20:00. Next-day booking typical.
Sample reaches the lab within 24 hours of collection.
Adults 16+ with photo ID. Paediatric draws by appointment at selected sites.
Mon–Fri, with Saturday hours at most metropolitan locations.
Samples processed same-day at the receiving clinic.
Every test is processed in a UKAS ISO 15189-accredited laboratory, overseen by GMC-registered physicians, and governed by UK GDPR. No overseas processing, no offshore data.
Follow these guidelines to ensure accurate, reproducible results. Most markers are sensitive to recent food, exercise, and sleep.
Can't find your answer? Our clinical support team is available Monday to Friday, 9am–5pm.
Contact supportHOMA-IR (Homeostatic Model Assessment of Insulin Resistance) is a calculated index derived from fasting glucose and fasting insulin. It estimates how much insulin the pancreas needs to secrete to maintain normal blood glucose, which is a proxy for insulin sensitivity. A high HOMA-IR means the body is resistant to insulin’s glucose-lowering signal and must compensate by secreting more. This is the core metabolic abnormality in type 2 diabetes and metabolic syndrome, and it typically develops ten to twenty years before glucose rises into the diabetic range. Addressing insulin resistance early through diet, exercise, and lifestyle changes is one of the most impactful interventions available for long-term health.
LDL cholesterol measures the mass of cholesterol carried by LDL particles but does not directly count the particles. ApoB measures each atherogenic lipoprotein particle directly because each particle — whether LDL, VLDL, IDL, or Lp(a) — contains exactly one ApoB molecule. In individuals with insulin resistance, small dense LDL particles are common; these have relatively little cholesterol per particle but are highly atherogenic. Such individuals can have a misleadingly normal or low LDL-C while carrying a large number of atherogenic particles, reflected in an elevated ApoB. Most cardiovascular research now indicates that ApoB is a superior predictor of coronary artery disease risk.
dehydroepiandrosterone sulphate (DHEA-S) (dehydroepiandrosterone sulphate) is the sulphated, circulating form of dehydroepiandrosterone (DHEA), a steroid hormone produced primarily by the adrenal glands. It serves as a precursor to both testosterone and oestrogen and peaks in early adulthood (typically the mid-twenties) then declines by approximately 2 percent per year. Low dehydroepiandrosterone sulphate (DHEA-S) relative to age-adjusted norms is associated with immune senescence, reduced muscle mass, increased cardiovascular risk, and cognitive decline. Some longevity medicine practitioners use dehydroepiandrosterone sulphate (DHEA-S) as a crude indicator of biological age relative to chronological age. It does not, however, definitively diagnose or predict any specific disease.
Annual testing is the most common recommendation for healthy adults over 35 who use preventative health monitoring as part of their lifestyle. The combination of markers in this panel captures the key metabolic, hormonal, and inflammatory trajectories that predict long-term health outcomes. Annual comparison allows you to detect unfavourable trends years before they become clinical problems. If you are actively modifying diet, exercise habits, medication, or supplementation, retesting after three to four months provides objective feedback on whether your interventions are working.
Yes, substantially and often within months. HbA1c responds to reduced carbohydrate intake and increased physical activity within eight to twelve weeks. Triglycerides fall rapidly with reduced alcohol and refined carbohydrate consumption. hs-CRP decreases with anti-inflammatory dietary patterns, improved sleep, and stress management. Testosterone in men responds to weight loss, resistance training, and improved sleep. Fasting insulin and HOMA-IR show some of the most dramatic improvements with low-carbohydrate diets and time-restricted eating. Retesting after a focused lifestyle intervention is a powerful motivational tool and a clinically meaningful health outcome measure.
Lipoprotein(a) — abbreviated Lp(a) — is a modified form of LDL with an additional protein (apo(a)) that makes it particularly atherogenic and thrombogenic. Unlike most cardiovascular risk factors, Lp(a) is almost entirely determined by genetics (the LPA gene) and does not respond meaningfully to diet, exercise, or standard cholesterol-lowering drugs such as statins. Elevated Lp(a) is a significant independent cardiovascular risk factor, estimated to affect around 20 percent of the population. If your Lp(a) is elevated, the appropriate response is cardiovascular risk reduction through other modifiable factors (blood pressure, smoking, lipids, glucose) and discussion with your GP about aspirin or emerging Lp(a)-lowering therapies.
